Progressivity of Variable Deceleration to Late Deceleration – A Case Report and It’s Implication
Abstract
Introduction: Cardiotocography (CTG) records changes in fetal heart rate and their temporal relationship with uterine contractions. This case report specifically highlights the progression of variable deceleration to late deceleration, its implication, and importance of variable deceleration.
Case Report: A 42-year-old G4P2A1 patient at 37–38 weeks of gestation presented to our emergency unit with severe preeclampsia (170/110 mmHg) and irregular heart rate (120 – 70 – 110 bpm). We performed CTG and showed baseline 120–130, with no variability and accompanied by deceleration. The first 2 deceleration occur without the same timing as contraction, and the two later occur after contraction. We performed C-section on the patient and healthy female baby.
Discussion: This case provides us with a rather unique pattern of CTG where we could see a slight progression from variable deceleration to late deceleration. Deceleration itself represents a reflex response of the fetus to reduce myocardial workload in response to stress; therefore, it can be secondary to cord compression or other causes. As this condition continues, the fetus deceleration progresses to late deceleration, presenting with a more dire condition and severe acidemic condition.
Conclusion: Most of the time variable deceleration are classified as “cord compression” decelerations, while most cases of fetal acidemia in labor are due to reduction in uteroplacental perfusion not the compression of cord. Therefore, variable deceleration is an important sign of fetal acidemia, and when such if itis present, we should take the initiative for termination of pregnancy to prevent bad outcomes of the fetus.
Progresivitas Deselerasi Variabel ke Deselerasi Lambat–Laporan Kasus dan Implikasinya
Abstrak
Pendahuluan:Kardiotokografi merekam perubahan detak jantung janin dan hubungannya dengan kontraksi uterus. Laporan kasus ini hendak menunjukkan perubahan dari deselerasi variabel ke deselerasi lambat serta implikasi dan pentingnya deselerasi variabel.
Laporan Kasus: Seorang wanita 42 tahun G4P2A1 gravida 37-38 minggu datang ke IGD dengan preeklamsia berat (170/110 mmHg) dan denyut jantung janin yang irreguler (120 – 70 – 110 x/menit). Setelah dilakukan kardiotokografi didapatkan baseline 120-130, tanpa ada akselerasi dan diikuti deselerasi. Dua deselerasi yang muncul pertama timbul tanpa ada hubungan dengan kontraksi uterus dan dua kontraksi berikut nya terjadi setelah kontraksi. Pasien kemudian dilakukan seksio sesarea dan lahir bayi perempuan sehat.
Diskusi: Kasus ini memberikan gambaran kardiotokografi unik dengan adanya progresivitas dari deselerasi variabel ke deselerasi lambat. Deselerasi sendiri merupakan respon fetus terhadap stress dengan menurunkan beban kerja myocardium janin. Hal ini terjadi akibat hipoksia pada janin yang terjadi akibat kompresi tali pusat dan atau penyebab lain. Bila kondisi ini berlanjut menjadi deselerasi lambat maka kondisi fetus akan semakin memburuk dan masuk kedalam asidemia berat.
Kesimpulan: Sering kali dalam melihat deselerasi variabel, kita menklasifikasikannya sebagai deselerasi yang disebabkan penekanan tali pusat/cord-compression yang bila dilakukan resusitasi dapat membaik. Akan tetapi mayoritas kasus dari asidemia fetus pada persalinan terjadi akibat penurunan aliran uteroplasental bukan dari kompresi tali pusat. Oleh karena itu deselerasi variabel justru merupakan tanda penting dalam menilai asidemia fetus dan bila ada dalam pemeriksaan karditokografi harus diambil langkah cepat untuk terminasi kehamilan guna mencegah luaran janin yang buruk.
Kata kunci : kardiotokografi; deselerasi variabel; asidemia fetus
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DOI: http://dx.doi.org/10.24198/obgynia.v7i1.640
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